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The Trans-NIH GeroScience Interest Group (GSIG) cordially invites you to its summer seminar, featuring Dr. Owen Wolkowitz. Dr. Wolkowitz is a Professor of Psychiatry at the University of California, San Francisco and Director of the Psychopharmacology Assessment Clinic. His teaching, clinical work and research are in the areas of stress, depression, and anxiety disorders. His interests include the effects of stress and stress hormones on the brain and behavior, as well as the identification of mechanisms underlying depression, which may lead to the discovery of new treatments. Dr. Wolkowitz and colleagues have developed a detailed model of mechanistic pathways leading from stress to psychiatric illness, representing effects of steroid and neurosteroid hormones, oxidative stress, neurotrophic factors, inflammatory responses, changes in telomeres and telomerase and genetic polymorphisms. They are now conducting additional studies to test this model and to explore the cumulative neuronal damage in the hippocampus.
The GeroScience Interest Group (GSIG) was formed to enhance opportunities for discussion of the intersection between the biology of aging and the biology of disease and conditions that are of interest across ICs. It is focused on basic biology, but with a longer view towards translation. If you are interested in learning more, please visit the GSIG web site http://sigs.nih.gov/geroscience/Pages/default.aspx
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NLM Title:
Getting old before our time : psychiatric illness and accelerated cell aging / Dr. Owen Wolkowitz.
Author:
Wolkowitz, Owen M. National Institutes of Health (U.S.). Geroscience Interest Group,
Publisher:
Abstract:
(CIT): Dr. Wolkowitz and colleagues have developed a detailed model of mechanistic pathways leading from stress to psychiatric illness, representing effects of steroid and neurosteroid hormones, oxidative stress, neurotrophic factors, inflammatory responses, changes in telomeres and telomerase and genetic polymorphisms. They are now conducting additional studies to test this model and to explore the cumulative neuronal damage in the hippocampus.
